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Pubmed ID :29931538
Publication Date : //

Alk5/Runx1 signaling mediated by extracellular vesicles promotes vascular repair in acute respiratory distress syndrome.


Pulmonary endothelial cells' (ECs) injury and apoptotic death are necessary and sufficient for the pathogenesis of the acute respiratory distress syndrome (ARDS), regardless of epithelial damage. Interaction of dysfunctional ECs with circulatory extracellular vesicles (EVs) holds therapeutic promise in ARDS. However, the presence in the blood of long-term ARDS survivors of EVs with a distinct phenotype compared to the EVs of non-surviving patients is not reported. With a multidisciplinary translational approach, we studied EVs from the blood of 33 patients with moderate-to-severe ARDS.

Authors : Shah Trushil , Qin Shanshan , Vashi Mona , Predescu Dan N , Jeganathan Niranjan , Bardita Cristina , Ganesh Balaji , diBartolo Salvatore , Fogg Louis F , Balk Robert A , Predescu Sanda A ,

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