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Pubmed ID :18507830
Publication Date : //

Could a defective epithelial sodium channel lead to bronchiectasis.


Bronchiectasis is defined as a permanent dilation of the airways arising from chronic bronchial inflammation/infection. In 50% of cases, no etiology can be identified. Recently, the role of the epithelial sodium channel ENaC has been pointed out in the pathophysiology of cystic fibrosis, a disease due to mutations in the CFTR gene and causing bronchiectasis in the airways. Moreover, it was found that transgenic mice overexpressing ENaCbeta present cystic fibrosis-like lung disease symptoms. Our aim was to evaluate if a defective ENaC protein could be involved in the development of bronchiectasis.

Authors : Fajac Isabelle , Viel Marion , Sublemontier Sébastien , Hubert Dominique , Bienvenu Thierry ,

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